安田 佳代

Yasuda Kayo

  • 講師
  • 学位:博士(医学)

基本情報

所属

  • Undergraduate School of Health Studies / Department of Health Management
  • Micro/Nano Technology Center

ジャンル

  • Lifestyle-Related Diseases

研究と関連するSDGs

  • Good Health and Well-Being

詳細情報

研究分野

  • Life sciences Cell biology
  • Life sciences Medical biochemistry

論文

Streptococcus thermophilus extends lifespan through activation of DAF-16-mediated antioxidant pathway in Caenorhabditis elegans.

Interaction between the ins/IGF-1 and p38 MAPK signaling pathways in molecular compensation of sod genes and modulation related to intracellular ROS levels in C. elegans.

Monitoring Age-Related Changes in the Lactate/Pyruvate Ratio Using a Colorimetric Assay in a C. elegans Model of Increased Life Span.

Metabolic Biomarkers in Nematode C. elegans During Aging.

Small-Scale Colorimetric Assays of Intracellular Lactate and Pyruvate in the Nematode Caenorhabditis elegans.

Impaired p53/CEP-1 is associated with lifespan extension through an age-related imbalance in the energy metabolism of C.elegans

Endogenous reactive oxygen species cause astrocyte defects and neuronal dysfunctions in the hippocampus: a new model for aging brain

Infertility and recurrent miscarriage with complex II deficiency-dependent mitochondrial oxidative stress in animal models

Oxidative Stress Induced Inflammation Initiates Functional Decline of Tear Production (vol 7, e45805, 2012)

Liposome-Encapsulated Hemoglobin Enhances Chemotherapy to Suppress Metastasis in Mice

Ethyl methanesulfonate induces mutations in Caenorhabditis elegans embryos at a high frequency

Genetically induced oxidative stress in mice causes thrombocytosis, splenomegaly and placental angiodysplasia that leads to recurrent abortion

Model animals for the study of oxidative stress from complex II

Model animals for the study of oxidative stress from complex II.

Oxidative Stress Induced Inflammation Initiates Functional Decline of Tear Production

Mitochondrial Superoxide Anion Overproduction in Tet-mev-1 Transgenic Mice Accelerates Age-Dependent Corneal Cell Dysfunctions

MRT letter: Expression of ATP sensor protein in Caenorhabditis elegans

A mutation in a mitochondrial dehydrogenase/reductase gene causes an increased sensitivity to oxidative stress and mitochondrial defects in the nematode Caenorhabditis elegans

Asymmetric Arginine Dimethylation Determines Life Span in C. elegans by Regulating Forkhead Transcription Factor DAF-16

Mitochondrial reactive oxygen species generation by the SDHC V69E mutation causes low birth weight and neonatal growth retardation

講演・口頭発表等

  • 抗老化を目指した人参湯類の漢方薬の評価
  • 抗老化を目指した生薬及び漢方薬の評価
  • 電子伝達系異常酸化ストレスによる自然免疫応答と大細胞肺がん
  • 抗老化を目指した生薬及び漢方薬の評価
  • 酸化ストレス依存的な自然免疫応答の慢性化を伴う非小細胞肺がんの発癌機序
  • 線虫C.elegansを用いた加齢とともに生じる個体差の解析
  • 酸化ストレス下における転写因子MXL‐3の機能
  • 線虫C.elegansのMXL‐3転写因子の酸化ストレス応答
  • センチュウ(C.エレガンス)による漢方薬の機能評価
  • 線虫における放射線ホルミシスと酸素効果
  • Type‐1 Fiber萎縮に関わる血流と遺伝子発現動態の解析と特徴
  • ミトコンドリア複合体II電子伝達異常に伴う組織間質への加齢依存的リンパ球浸潤の亢進
  • 酸化ストレスと老化のモデル研究
  • 線虫C.elegansのMXL‐3転写因子の酸化ストレス応答
  • 造腫瘍性SDHC変異モデルマウスでの加齢依存的炎症応答の解析
  • ミトコンドリア電子伝達系傷害に伴う慢性的酸化ストレス負荷による脳内グリア環境の加齢変化
  • 廃用性筋萎縮モデルにおける遺伝子発現の経時的変化と血流動態との関係
  • ミトコンドリア複合体II電子伝達異常に起因する酸化ストレスと生体の加齢変容

所属学会

  • THE MOLECULAR BIOLOGY SOCIETY OF JAPAN
  • JAPAN SOCIETY FOR BIOMEDICAL GERONTOLOGY

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