中尾 周

Nakao Shu

  • 准教授
  • 学位:博士(獣医学)

基本情報

所属

  • Undergraduate School of Medicine / Faculty of Medicine

詳細情報

研究キーワード

  • Cardiac Physiology
  • Histology
  • Molecular Biology
  • Biochemistry
  • Cell Biology
  • Ion channel
  • Calcium
  • Veterinary Medine
  • Cardiac conduction system
  • Cardiomyocytes
  • Cardiac hypertrophy
  • Arrhythmias
  • Cardiology

研究分野

  • Life sciences Veterinary medicine Veterinary Pathology
  • Life sciences Veterinary medicine Veterinary Cardiology
  • Life sciences Cardiology Cardiac arrhythmias
  • Life sciences Physiology

受賞

  • Physiological Society of Japan The 23rd Promotion Award of the Physiological Society of Japan for Young Scientists
  • ISCP Oral Presentation Award
  • JSVC The 108th JSVC Best Presentation Awards
  • ISCP 23rd ISCP Best Abstract Award (3rd prize)
  • Naito Foundation Fellowships for study abroad
  • Nakatomi Foundation Fellowships for study abroad

論文

c-Myc/microRNA-17-92 Axis Phase-Dependently Regulates PTEN and p21 Expression via ceRNA during Reprogramming to Mouse Pluripotent Stem Cells

Canonical Wnt signaling activation by chimeric antigen receptors for efficient cardiac differentiation from mouse embryonic stem cells.

Remodeling of the Purkinje Network in Congestive Heart Failure in the Rabbit

Regulation of sinus node pacemaking and atrioventricular node conduction by HCN channels in health and disease.

Intrinsic Electrical Remodeling Underlies Atrioventricular Block in Athletes

ETS-dependent enhancers for endothelial-specific expression of serum/glucocorticoid-regulated kinase 1 during mouse embryo development.

Common arterial trunk in a cat: a high-resolution morphological analysis with micro-computed tomography

Pathological Features of the Atrioventricular Conduction System in Feline Cases of Third-degree Atrioventricular Block

Molecular Basis of Cardiac Impulse Generation in Sinoatrial Node in Health and Disease

Silencing miR-370-3p rescues funny current and sinus node function in heart failure

Chimeric G-CSF Receptor-Mediated STAT3 Activation Contributes to Efficient Induction of Cardiomyocytes from Mouse Induced Pluripotent Stem Cells

STAT3 for cardiac regenerative medicine: Involvement in stem cell biology, pathophysiology, and bioengineering

Applications for induced pluripotent stem cells in disease modelling and drug development for heart diseases

Metabolic remodeling during somatic cell reprogramming to induced pluripotent stem cells: Involvement of hypoxia-inducible factor 1

Emerging roles of neuronal Ca2+ sensor-1 in cardiac and neuronal tissues: A mini review

Serum/glucocorticoid-regulated kinase 1 as a novel transcriptional target of bone morphogenetic protein-ALK1 receptor signaling in vascular endothelial cells

Overexpression of heart-specific small subunit of myosin light chain phosphatase results in heart failure and conduction disturbance

Targeting miR-423-5p Reverses Exercise Training-Induced HCN4 Channel Remodeling and Sinus Bradycardia.

講演・口頭発表等

  • 長期持久性運動における心ブロックの分子的基盤(Molecular Basis of Heart Block in Chronic Endurance Exercise)

所属学会

  • JAPANESE SOCIETY OF VETERINARY CARDIOLOGY
  • JAPANESE HEART RHYTHM SOCIETY
  • THE PHYSIOLOGICAL SOCIETY OF JAPAN
  • THE PHYSIOLOGICAL SOCIETY
  • Japanese Society of Cardiology

共同研究・競争的資金等の研究課題

Metabolic remodeling in dysfunction of the cardiac conduction system: a role of miRNAs

Investigation of key molecules for exercise-induced bradycardia: involvement of oxidative stress in functional regulation of the cardiac pacemaker

Development of direct reprogramming into cardiac pacemaker cells for a novel regenerative medicine

Novel mechanism for energy metabolism regulated by a Ca2+-sensor protein

A role of neuronal calcium sensor-1 on the nuclear calcium regulation in cardiomyocytes: contribution to cardiac hypertrophy

Molecular mechanism for regulaiton of ion transporter via lipid signaling and its structural basis

ResearchMapへ移動します

Contact Us

Inquiries about coverage

Public Affairs Division Public Affairs and Communications Department

Tel. 0463-63-4670(direct dialing)